Increased Serum Igg In Adult

Adult Bovine Serum - 500 ml

CAABS-1A each
EUR 52.4

Igg Serum Laboratories manufactures the increased serum igg in adult reagents distributed by Genprice. The Increased Serum Igg In Adult reagent is RUO (Research Use Only) to test human serum or cell culture lab samples. To purchase these products, for the MSDS, Data Sheet, protocol, storage conditions/temperature or for the concentration, please contact IgG Serum. Other Increased products are available in stock. Specificity: Increased Category: Serum Group: Igg In

*Mouse IgG Depleted Serum

each
EUR 3730
Description: *Mouse IgG Depleted Serum

*Mouse IgG Depleted Serum

each
EUR 466
Description: *Mouse IgG Depleted Serum

*Rat IgG Depleted Serum

each
EUR 427
Description: *Rat IgG Depleted Serum

*Rat IgG Depleted Serum

each
EUR 1377
Description: *Rat IgG Depleted Serum

Echinococcus IgG Control Serum

3 mL
EUR 105

Tetanus IgG Control Serum

3 mL
EUR 105

Cytomegalovirus IgG Control Serum

3 mL
EUR 105

Igg In information

PMS2L5, CT (PMS2P5, PMS2L5, PMS4, PMS7, Postmeiotic segregation increased 2-like protein 5, Postmeiotic segregation increased protein 4, Postmeiotic segregation increased protein 7, Putative postmeiotic segregation increased 2 pseudogene 5) (MaxLight 650)

MBS6335232-5x01mL 5x0.1mL
EUR 4250

PMS2L5, CT (PMS2P5, PMS2L5, PMS4, PMS7, Postmeiotic segregation increased 2-like protein 5, Postmeiotic segregation increased protein 4, Postmeiotic segregation increased protein 7, Putative postmeiotic segregation increased 2 pseudogene 5) (MaxLight 750)

MBS6335233-01mL 0.1mL
EUR 980

PMS2L5, CT (PMS2P5, PMS2L5, PMS4, PMS7, Postmeiotic segregation increased 2-like protein 5, Postmeiotic segregation increased protein 4, Postmeiotic segregation increased protein 7, Putative postmeiotic segregation increased 2 pseudogene 5) (MaxLight 750)

MBS6335233-5x01mL 5x0.1mL
EUR 4250

PMS2 (Postmeiotic Segregation Increased 2)

MO47015 100 ul
EUR 418.8

PMS2 (Postmeiotic Segregation Increased 2)

MBS556114-01mL 0.1mL
EUR 455

PMS2 (Postmeiotic Segregation Increased 2)

MBS556114-5x01mL 5x0.1mL
EUR 1895

PMS2 Antibody / Post Meiotic Segregation Increased 2

V5202-100UG 100ug
EUR 363.3
Description: PMS2 is involved in DNA mismatch repair. It forms a heterodimer with MLH1 and this complex interacts with other complexes bound to mismatched bases. Defects in PMS2 are the cause of hereditary non-polyposis colorectal cancer type 4 (HNPCC4). Mutations in more than one gene locus can be involved alone or in combination in the production of the HNPCC phenotype (also called Lynch syndrome). Most families with clinically recognized HNPCC have mutations in either MLH1 or MSH2 genes. HNPCC is an autosomal, dominantly inherited disease associated with marked increase in cancer susceptibility. It is characterized by a familial predisposition to early onset colorectal carcinoma (CRC) and extra-colonic cancers of the gastrointestinal, urological and female reproductive tracts. HNPCC is reported to be the most common form of inherited colorectal cancer in the Western world, and accounts for 15% of all colon cancers. Defects in PMS2 are a cause of mismatch repair cancer syndrome (MMRCS); also known as Turcot syndrome or brain tumor-polyposis syndrome 1 (BTPS1). MMRCS is an autosomal dominant disorder characterized by malignant tumors of the brain associated with multiple colorectal adenomas. Skin features include sebaceous cysts, hyperpigmented and cafe au lait spots.

PMS2 Antibody / Post Meiotic Segregation Increased 2

V5202-20UG 20ug
EUR 160.3
Description: PMS2 is involved in DNA mismatch repair. It forms a heterodimer with MLH1 and this complex interacts with other complexes bound to mismatched bases. Defects in PMS2 are the cause of hereditary non-polyposis colorectal cancer type 4 (HNPCC4). Mutations in more than one gene locus can be involved alone or in combination in the production of the HNPCC phenotype (also called Lynch syndrome). Most families with clinically recognized HNPCC have mutations in either MLH1 or MSH2 genes. HNPCC is an autosomal, dominantly inherited disease associated with marked increase in cancer susceptibility. It is characterized by a familial predisposition to early onset colorectal carcinoma (CRC) and extra-colonic cancers of the gastrointestinal, urological and female reproductive tracts. HNPCC is reported to be the most common form of inherited colorectal cancer in the Western world, and accounts for 15% of all colon cancers. Defects in PMS2 are a cause of mismatch repair cancer syndrome (MMRCS); also known as Turcot syndrome or brain tumor-polyposis syndrome 1 (BTPS1). MMRCS is an autosomal dominant disorder characterized by malignant tumors of the brain associated with multiple colorectal adenomas. Skin features include sebaceous cysts, hyperpigmented and cafe au lait spots.

PMS2 Antibody / Post Meiotic Segregation Increased 2

V5202SAF-100UG 100ug
EUR 363.3
Description: PMS2 is involved in DNA mismatch repair. It forms a heterodimer with MLH1 and this complex interacts with other complexes bound to mismatched bases. Defects in PMS2 are the cause of hereditary non-polyposis colorectal cancer type 4 (HNPCC4). Mutations in more than one gene locus can be involved alone or in combination in the production of the HNPCC phenotype (also called Lynch syndrome). Most families with clinically recognized HNPCC have mutations in either MLH1 or MSH2 genes. HNPCC is an autosomal, dominantly inherited disease associated with marked increase in cancer susceptibility. It is characterized by a familial predisposition to early onset colorectal carcinoma (CRC) and extra-colonic cancers of the gastrointestinal, urological and female reproductive tracts. HNPCC is reported to be the most common form of inherited colorectal cancer in the Western world, and accounts for 15% of all colon cancers. Defects in PMS2 are a cause of mismatch repair cancer syndrome (MMRCS); also known as Turcot syndrome or brain tumor-polyposis syndrome 1 (BTPS1). MMRCS is an autosomal dominant disorder characterized by malignant tumors of the brain associated with multiple colorectal adenomas. Skin features include sebaceous cysts, hyperpigmented and cafe au lait spots.

PMS2 Antibody / Post Meiotic Segregation Increased 2

V5203-100UG 100ug
EUR 363.3
Description: PMS2 is involved in DNA mismatch repair. It forms a heterodimer with MLH1 and this complex interacts with other complexes bound to mismatched bases. Defects in PMS2 are the cause of hereditary non-polyposis colorectal cancer type 4 (HNPCC4). Mutations in more than one gene locus can be involved alone or in combination in the production of the HNPCC phenotype (also called Lynch syndrome). Most families with clinically recognized HNPCC have mutations in either MLH1 or MSH2 genes. HNPCC is an autosomal, dominantly inherited disease associated with marked increase in cancer susceptibility. It is characterized by a familial predisposition to early onset colorectal carcinoma (CRC) and extra-colonic cancers of the gastrointestinal, urological and female reproductive tracts. HNPCC is reported to be the most common form of inherited colorectal cancer in the Western world, and accounts for 15% of all colon cancers. Defects in PMS2 are a cause of mismatch repair cancer syndrome (MMRCS); also known as Turcot syndrome or brain tumor-polyposis syndrome 1 (BTPS1). MMRCS is an autosomal dominant disorder characterized by malignant tumors of the brain associated with multiple colorectal adenomas. Skin features include sebaceous cysts, hyperpigmented and cafe au lait spots.

PMS2 Antibody / Post Meiotic Segregation Increased 2

V5203-20UG 20ug
EUR 160.3
Description: PMS2 is involved in DNA mismatch repair. It forms a heterodimer with MLH1 and this complex interacts with other complexes bound to mismatched bases. Defects in PMS2 are the cause of hereditary non-polyposis colorectal cancer type 4 (HNPCC4). Mutations in more than one gene locus can be involved alone or in combination in the production of the HNPCC phenotype (also called Lynch syndrome). Most families with clinically recognized HNPCC have mutations in either MLH1 or MSH2 genes. HNPCC is an autosomal, dominantly inherited disease associated with marked increase in cancer susceptibility. It is characterized by a familial predisposition to early onset colorectal carcinoma (CRC) and extra-colonic cancers of the gastrointestinal, urological and female reproductive tracts. HNPCC is reported to be the most common form of inherited colorectal cancer in the Western world, and accounts for 15% of all colon cancers. Defects in PMS2 are a cause of mismatch repair cancer syndrome (MMRCS); also known as Turcot syndrome or brain tumor-polyposis syndrome 1 (BTPS1). MMRCS is an autosomal dominant disorder characterized by malignant tumors of the brain associated with multiple colorectal adenomas. Skin features include sebaceous cysts, hyperpigmented and cafe au lait spots.

PMS2 Antibody / Post Meiotic Segregation Increased 2

V5203SAF-100UG 100ug
EUR 363.3
Description: PMS2 is involved in DNA mismatch repair. It forms a heterodimer with MLH1 and this complex interacts with other complexes bound to mismatched bases. Defects in PMS2 are the cause of hereditary non-polyposis colorectal cancer type 4 (HNPCC4). Mutations in more than one gene locus can be involved alone or in combination in the production of the HNPCC phenotype (also called Lynch syndrome). Most families with clinically recognized HNPCC have mutations in either MLH1 or MSH2 genes. HNPCC is an autosomal, dominantly inherited disease associated with marked increase in cancer susceptibility. It is characterized by a familial predisposition to early onset colorectal carcinoma (CRC) and extra-colonic cancers of the gastrointestinal, urological and female reproductive tracts. HNPCC is reported to be the most common form of inherited colorectal cancer in the Western world, and accounts for 15% of all colon cancers. Defects in PMS2 are a cause of mismatch repair cancer syndrome (MMRCS); also known as Turcot syndrome or brain tumor-polyposis syndrome 1 (BTPS1). MMRCS is an autosomal dominant disorder characterized by malignant tumors of the brain associated with multiple colorectal adenomas. Skin features include sebaceous cysts, hyperpigmented and cafe au lait spots.

Postmeiotic Segregation Increased 2 (PMS2) (PT2116) Antibody

N1752-100uL 100uL
EUR 122.5
Description: Human Postmeiotic Segregation Increased 2 (PMS2) (PT2116) Mouse Monoclonal Antibody

Postmeiotic Segregation Increased 2 (PMS2) (PT2116) Antibody

N1752-50uL 50uL
EUR 66.5
Description: Human Postmeiotic Segregation Increased 2 (PMS2) (PT2116) Mouse Monoclonal Antibody

Postmeiotic Segregation Increased 2-Like Protein 5 (PMS2L5) Antibody

abx030611-400ul 400 ul
EUR 627.6

Postmeiotic Segregation Increased 2-Like Protein 5 (PMS2L5) Antibody

abx030611-80l 80 µl
EUR 343.2

Postmeiotic Segregation Increased 2-Like Protein 5 (PMS2L5) Antibody

abx030611-400l 400 µl
EUR 518.75